A patient presents with symptoms of chronic liver disease and increased blood glucose. Which complication may arise?

In the context of chronic liver disease, portal hypertension is a significant complication that arises due to the liver's inability to properly process blood from the portal vein. Chronic liver diseases, such as cirrhosis, lead to structural changes and fibrosis within the liver, causing increased resistance to blood flow. This elevated resistance ultimately results in increased pressure within the portal venous system, leading to portal hypertension.

Portal hypertension can have various clinical manifestations, including the development of varices (enlarged veins) in the esophagus and stomach, which can rupture and cause life-threatening hemorrhage. Patients may also experience splenomegaly (enlargement of the spleen) and ascites, resulting from increased pressure and altered fluid dynamics in the body.

The presence of increased blood glucose in conjunction with chronic liver disease suggests that liver function is impaired, potentially affecting insulin metabolism and glucose homeostasis. In this context, the development of portal hypertension as a complication aligns with the physiological changes occurring in chronic liver disease.

While acute renal failure, non-alcoholic fatty liver disease, and cholecystitis are conditions that can be associated with various complications, they do not directly stem from the pathophysiological changes of chronic liver disease in the same way